COVID vaccine inequity, species swaps — the week in infographics

来自：
https://www.nature.com/articles/d41586-021-02151-z

COVID vaccine inequity, species swaps — the week in infographics

COVID疫苗不充足，种类的交换——一周内的信息图

Inequity in vaccine access

Rich nations’ plans to administer booster doses of COVID-19 vaccine to people who have been fully vaccinated have drawn criticism from many global health researchers, who highlight the growing disparities between wealth and access to vaccines.
A July report from KFF, a health-policy organization based in San Francisco, California, finds that at current vaccination rates, low-income countries won’t achieve substantial levels of protection until at least 2023.

疫苗获取的不充足
富有国家的计划——推进人们全面接种COVID-19疫苗，收到来自许多健康组织的批评，他们指出这拉大了不同国家的疫苗的接种率（充足与有权接种疫苗）
来自KFF的报告指出……

The changing face of ecosystems

Despite alarming declines in some animal and plant species, total biodiversity in many ecosystems is not decreasing. But that doesn’t mean such ecosystems are static. In fact, the mix of species in local communities is changing rapidly almost everywhere on Earth. As some inhabitants disappear, colonizers move in and add to species richness.

生态系统的改变

尽管某些动物和植物处于警觉的下降状态，总的生物多样性在很多生态系统是没有减少的。但这不意味着这样的生态系统是静止的。事实上，物种在当地社区的混合在地球上是在急速地变化。随着某些原住民的消失，殖民者会进驻从而丰富物种的多样性。

Biodiversity—species richness

Genetics behind the menopause

Genetic variants associated with age at onset of menopause have been identified in a large-scale genomic analysis, findings that bring scientists a step closer to predicting and treating early menopause.
When the DNA of egg cells in ovaries is damaged in mice, expression of the gene Chek1 promotes DNA repair, whereas expression of Chek2 promotes destruction of the affected cell.
The analysis found that variants of the human equivalent of Chek2 and other genes involved in the response to DNA damage are associated with differences in age at natural menopause. It also showed that mice carrying an extra copy of Chek1, or lacking expression of Chek2, had a longer reproductive age span than did typical mice.
基因变异（与绝经期开始相关联的）被确认是在一个大规模的基因组分析之中，其中的发现使得科学家更进一步去预测和治疗绝经。
当老鼠的DNA的卵细胞被毁坏，Chek1基因的表达促使DNA修复，相反，Chek2的表达加速病变细胞的毁灭。
分析发现，变异在人类等同的Chek2基因以及其他涉及DNA毁灭反应在内的基因是与（在自然的绝经中）年龄的差异相关的这也表明老鼠携带一个额外的Chek1复制，或者缺乏Chek2基因的表达，它有一个更长的生殖年龄。